The '2045 Initiative' Says Immortality Will Come Via Brain Computers And Hologram Bodies In 30 Years

Want to be immortal? A Russian group called the 2045 Initiative has a plan, and they hope to make it a reality during our lifetimes. From their website:

"The main objectives of the Initiative are: the creation of a new vision of human development that meets global challenges humanity faces today, realization of the possibility of a radical extension of human life by means of cybernetic technology, as well as the formation of a new culture associated with these technologies."

These "cybernetic technologies" take many forms. In the end the group, founded by Dmitry Itskov in February of 2011, think humans can attain immortality not by outsmarting disease and pathogens, but by harnessing nanotechnology, robots and brain-computer interfaces to create hologram bodies with artificial brains that can be programmed with your own personality.

Oh, by the way, this not only requires intense investigation in pretty much every area of science, but also involves a complete overhaul of our social, ethical and pretty much every other part of our human lives. Yeah. That will happen by 2045. Good job setting attainable goals, guys.
Here's the plan for holographic immortality if you want to get on board:

2012-2013. The global economic and social crises are exacerbated. The debates on the global paradigm of future development intensifies.

New transhumanist movements and parties emerge. Russia 2045 transforms into World 2045.

Simultaneously, the 2045.com international social network for open innovation is expanding. Here anyone interested may propose a project, take part in working on it, or fund it, or both. In the network, there are scientists, scholars, researchers, financiers and managers.

2013-2014. New centers working on cybernetic technologies for the development of radical life extension rise. The 'race for immortality' starts.

2015-2020. The Avatar is created — A robotic human copy controlled by thought via 'brain-computer' interface. It becomes as popular as a car.

2020. In Russia and in the world appear — in testing mode — several breakthrough projects:
Android robots replace people in manufacturing tasks; android robot servants for every home; thought-controlled Avatars to provide telepresence in any place of the world and abolish the need business trips; flying cars; thought driven mobile communications built into the body or sprayed onto the skin.

2020-2025. An autonomous system providing life support for the brain and allowing it interaction with the environment is created. The brain is transplanted into an Avatar B. With Avatar B man receives new, expanded life.

2025. The new generation of Avatars provides complete transmission of sensations from all five sensory robot organs to the operator.

2030-2035. ReBrain — The colossal project of brain reverse engineering is implemented. World science comes very close to understanding the principles of consciousness.

2035. The first successful attempt to transfer one's personality to an alternative carrier. The epoch of cybernetic immortality begins.

2040-2050. Bodies made of nanorobots that can take any shape arise alongside hologram bodies.

2045-2050. Drastic changes in social structure, and in scientific and technological development. All the
for space expansion are established.
For the man of the future, war and violence are unacceptable. The main priority of his development is spiritual self-improvement.

A new era dawns: The era of neohumanity.

See the video below if you want your brains scrambled. It seriously looks and sounds like something from a science fiction movie. Anyone know if this is a hoax? Or a publicity stunt for some movie? I really can't get my brain around it. I don't know what world they are living on, but it's definitely not the same one we are.


Learn more about or participate in the 2045 project  

Original post by business insider

Cryogenics Humans

If you are interested in Cryogenics like me then I found a great video. Its a short documentary about 18mins long on the process of Cryogenics and Cryonics.

"Dying for Immortality"

Aging is recorded in our genes

Researchers at the Bellvitge Biomedical Research Institute in Barcelona, Spain have found a significantly higher amount of cytosine methylation in the newborn than in the centenarian: 80.5% of all cytosine nucleotides, compared with 73%.

Recent research suggests that changes in DNA methylation patterns as a person gets older may contribute to human diseases for which risk increases with age, including cancer.

Background:

DNA is made up of four basic building blocks — adenine, thymine, guanine, and cytosine—and the sequence of these nucleotides within a gene determines what protein it makes. Genes can be switched on and off as needed, and the regulation of genes often involves what are called epigenetic mechanisms in which chemical alterations are made to the DNA. One of the most common of these epigenetic changes involves a methyl group — one carbon atom and three hydrogen atoms — binding to a nucleotide, usually cytosine. In general, this binding, called methylation, turns off the gene in question.

The researchers looked at two extreme cases: A newborn male baby and a man aged 103 years.

The team extracted DNA from white blood cells taken from the blood of the elderly man and from the umbilical cord blood of the baby and determined its methylation pattern using a fairly new technique called whole-genome bisulfite sequencing (WGBS).

With WGBS, DNA is exposed to the chemical sodium bisulfite, which has no effect on cytosines with methyl groups bound to them but turns nonmethylated cytosines into another nucleotide called uracil. The result is an epigenetic map that shows exactly which DNA sites are methylated and which are not.

To look at an intermediate case, the team also performed WGBS on the DNA of a 26-year-old male subject; the methylation level was also intermediate, about 78%.

Differences between newborn and centenarian

They then took a closer look at the differences between the DNA of the newborn and of the centenarian, but restricted the comparison to regions of the genome where the DNA nucleotide sequences were identical so that only the epigenetic differences would stand out.

The team identified nearly 18,000 “differentially methylated regions” (DMRs) of the genome, covering many types of genes. More than a third of the DMRs occurred in genes that have already been linked with cancer risk. Moreover, in the centenarian, 87% of the DMRs involved the loss of the methyl group, while only 13% involved the gain of one.

Finally, to expand the study, the team looked at the methylation patterns of 19 newborns and 19 people aged between 89 and 100 years old. This analysis confirmed that older people have a lower amount of cytosine methylation than newborns.

Increased risk of infection and diabetes

The authors conclude that the degree of methylation decreases in a cumulative fashion over time. Moreover, in the centenarian, the loss of methyl groups, which turns genes back on, often occurred in genes that increase the risk of infection and diabetes when they are turned on during adulthood. In contrast, the small number of genes in the centenarian that had greater methylation levels were often those that needed to be kept turned on to protect against cancer.

The new work is the first to compare the complete, genome-wide DNA methylation patterns of these two diverse age groups, says Martin Widschwendter, an oncologist at University College London in the United Kingdom who has studied the link between methylation and cancer.

Widschwendter, who likens the DNA sequence to the genome’s “hardware” and epigenetic changes to its “software,” says that the team’s study supports earlier research suggesting that “as a function of age and environmental exposure, this software accumulates defects” that can cause “age-related cancer and degenerative diseases.”

Ref.: Holger Heyn et al., Distinct DNA methylomes of newborns and centenarians, Proceedings of the National Academy of Sciences, 2012, DOI: 10.1073/pnas.1120658109 (open access)

[ Science Now ]